Stepby step, protocols are rising that will allow real functionality tobe restored for aging humanity. This one is promising because ayouthful blood system will surely cleanse the whole biological systemand plausibly allow healthy progress into the second century. It isnot likely to get much past that mark, but somewhat healthy andenergetic is something the wild achieves all the time.
We are entering a perfect storm of aging populations and rapidlyemerging anti aging protocols to extend those lives and even torestore general functionality. The level of change is alreadyastonishing and about to become more urgent as the public wakes up topossibilities and the desirability of an engaged older population.
Because I expected this decades ago due to my understanding of thenature of the knowledge growth curve, I never took mandatoryretirement particularly seriously. Now we have seen that eliminatedhere at least. All things been equal it is now plausible for someonetoday aged sixty five to still be fully engaged into his nineties. More important though is that he will not be alone.
Longevitygene points to 'fountain of youth'
A "longevitygene" can help boost the production of blood cells by reversingthe decline of stem cells in old age, a study suggests.
By NickCollins, Science Correspondent
6:34PM GMT 31 Jan 2013
http://www.telegraph.co.uk/science/science-news/9840532/Longevity-gene-points-to-fountain-of-youth.html
Blood-generating stemcells become slower in old age, increasing stress on the body, butinfusing them with a gene linked to the ageing process can restorethem to a more youthful state, researchers found.
The study, which wasperformed in mice, raises the possibility of new treatments fordegenerative diseases which occur with age, such as Alzheimer's.
More research will beneeded to determine whether the findings in mice also apply tohumans, but scientists claimed they could lead to the development ofa "molecular fountain of youth".
Scientists from theUniversity of California, Berkeley found that SIRT3, a proteinfrom a class known as sirtuins which has previously been linked toageing, could actually help "reverse" the natural process.
They examined thefunctioning of blood stem cells, which help keep the blood systemhealthy, on mice which had lacked the gene responsible for producingSIRT3.
The study, publishedin the Cell Reports journal, showed that for young mice theabsence of SIRT3 made no difference, but older mice without the genehad fewer blood stem cells and were less able to produce new bloodcells.
This could be becauseolder mice were more vulnerable to "oxidative stress", aharmful side effect of the metabolism, and needed their antioxidantsystem, which is boosted by SIRT3, to counteract it, researcherssaid.
When levels of SIRT3were raised in older mice, their blood cells were rejuvenated andproduction of blood cells improved.
Dr Danica Chen, wholed the study, said: "We already know that sirtuins regulateageing, but our study is really the first one demonstrating thatsirtuins can reverse ageing-associated degeneration, and I thinkthat's very exciting.
"This opens thedoor to potential treatments for age-related degenerative diseases... the question is whether we can understand the process well enoughso that we can actually develop a molecular fountain of youth."
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